Most of the site will reflect the ongoing surgical activity of Prof. Munir Elias MD., PhD. with brief slides and weekly activity. For reference to the academic and theoretical part, you are welcome to visit
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16-DECEMBER-2012 SIHAM MUHAMED FATTAH 52 YEARS
EXTRUDED DISC C5-6 WITH SECONDARY STENOSIS.
Anamnesis
The patient came to the clinic 01-November-2012
complaining of neck pain for 25 years with
exacerbation the last year with both shoulder
pain more the right. She has also LBP with left
sciatica for 4 years. She has urinary problems
for 4 years. MRI of the left knee done
11-October-2012 showing effusion of the left
knee.
On examination: the patient
is limping due to left sciatica with exaggerated
scoliotic stance. There is weak grip and
extension of the right hand and the right
triceps muscle 3/5. There was hypalgesia of the
thumb of the right hand. SLRS was 90 degrees
both sides without pain and there is weak
dorsiflexion left foot 4/5.
MRI of the cervical spine done
04-November-2012 showing wide-based extrusion of
C5-6 with bulge disci C4-5 and C6-7. MRI lumbar
spine showed small extruded disc L5-S1 more to
the left.
The patient then came
15-December-2012 complaining of both upper limb
pain with neck pain when looking to all
directions with weak both upper limbs. The
patient was seen by cardiologist, confirming
there was no cardiac problems. The left sciatica
increase the last 2 weeks and new MRI of the
lumbar spine was the same as before.
The patient was sent for new
MRI of the cervical spine which was done the
same day and it was the same as before.
Discectomy of C5-6 with
removal of the extrusion until the dura was seen
at all the posterior aspect. A Fidji cervical
cage 12x15x5.3 mm was inserted to the disc space
with NovaBone Putty CMP. Using cervical plate
Zimmer 22 mm one level with 4 fixed screws
4.2x14 mm, fusion of C5-6 was achieved. All
steps were done under C-arm control.
Routine closure of the wound.
The power of the upper limbs became normal.
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Comments
The patient has wide-based
extrusion of C5-6 with secondary canal stenosis.
There is no malacia but irritation of the
anterior spinal artery could provoke the
clinical data of myelopathy without the presence
of malacia.
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